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partially mediated via their aromatization to estrogens [108] In line with the latter observation BarettConnor et al observed a signi cant negative and graded association between levels of total and bioavailable estradiol, but not of BT and fracture prevalence in males (median age 67 years, range 56 87 years) independently of age, body mass index, or exercise [109] While the evidence for a signi cant role of estrogens in bone health is growing, the effects of T should not be dismissed Levels of BT correlated with all regions of proximal femur BMD and total body BMD after adjustment for age [92] It is to be remembered that estrogens are largely (>80%) products of peripheral aromatization of androgens In sum, the relevance of androgens (both in their own right and as precursors for the formation of estrogens) in age-associated osteopenia seems clear on the basis of these recent large-scale studies However the extent of the effects of T therapy is limited with respect to what has been reported in young hypogonadal males how to create pdf in javafx Generate Simple PDF Report using Jasper Report and Java ...
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In this iText tutorial, I am writing various code examples read a pdf file and generate PDF file. iText library helps to generate pdf files from java applications ... Premenopausal women, in comparison to men, are felt to be protected against cardiovascular disease by estrogens It is then paradoxical that in cross-sectional studies of men, elevated levels of estrogens [115] and relatively low levels of T [116,122,123] appear to be associated with coronary disease and myocardial infarction These studies suggest the intriguing possibility that, in spite of the prior assumption of overall negative effects of androgens on lipid pro les, a lower-than-normal androgen level in aging men is associated with an increase of atherosclerotic disease In a study of geriatric male patients who had suffered a myocardial infarction, it was found that these patients had low T levels [116] In addition four trials have found that pharmacologic doses of T relax coronary arteries when injected intraluminally, and modest but consistent improvement of exercise-induced angina and electrocardiographic ischemia have been observed following short-term T administration in at least ve RCTs [123 127] These results are encouraging but they await longer-term investigations On the other hand no bene cial effect could be demonstrated on peripheral arterial disease, and possible effects on cerebrovascular disease have not been evaluated [124,126] The apparently negative effect of androgens in old studies may have been due, in part, to the supraphysiologic levels of T achieved when injectable T esters were used The explanation may also lie in the fact that T also exerts both bene cial and adverse effects on the vascular wall, and that a complex of risk factors for cardiovascular disease termed syndrome X or the metabolic syndrome (comprising hypertension, insulin resistance, hypertriglyceridemia, and visceral obesity) is associated with low T levels [83,84,115,128] create pdf from jsp example Java PDF Libraries - Java Code Geeks
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Validate PDF files against the PDF/A-1b standard. Print. Print a PDF file using the standard Java printing API. Save as Image. Save PDFs as image files, such as PNG or JPEG. Create PDFs. Create a PDF from scratch, with embedded fonts and images. Signing. Digitally sign PDF files. . Premenopausal women suffer signi cantly less from cardiovascular disease than men, and traditionally it has been thought that the relationship between sex steroids and cardiovascular disease is predominantly determined by the relatively bene cial effects of estrogens, and by the relatively detrimental effects of androgens on lipid pro les [101] Nevertheless, the vast majority of cross-sectional studies in men are not in agreement with this assumption; they show a positive correlation between FT levels and HDL cholesterol [111 113], and a negative correlation, with brinogen, plasminogen activator inhibitor-1 [105], and insulin levels as well as with coronary heart disease [115,116], but not with cardiovascular mortality [117 119] Recent research shows the effects of sex steroids on biologic systems other than lipids Fat distribution, endocrine/paracrine factors produced by the vascular wall (such as endothelins, nitric oxide), blood platelets and coagulation, must also be considered in the analysis of the relationship between sex steroids and cardiovascular disease [120,121] 63 64 Vascular cells contain T and estradiol (E2) receptors as well as converting enzymes needed for E2 production Therefore T may regulate vascular physiology, directly or indirectly [126] It may especially modulate vascular reactivity by both endotheliumdependent and -independent mechanisms Physiologic doses reduce vasodilation through genomic actions As already described, supraphysiologic Hormones, Metabolism, Aging, and Men s Health doses enhance vasodilation through non-genomic action In addition, T exerts both pro-atherogenetic (rapid accumulation) and anti-atherogenetic effects (lipid secretion) on macrophage functions best pdf generation library java how to generate the pdf report from jsp - RoseIndia
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